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MatchTypeWhy
Tumor EscapeConcept Why?
Kohler, MarkPerson Why?
The combination of anti-KIR monoclonal antibodies with anti-PD-1/PD-L1 monoclonal antibodies could be a critical breakthrough in overcoming tumor immune escape in NSCLC.Academic Article Why?
Adoptively transferred immune T cells eradicate established tumors despite cancer-induced immune suppression.Academic Article Why?
C reactive protein impairs adaptive immunity in immune cells of patients with melanoma.Academic Article Why?
Deficiency of activated STAT1 in head and neck cancer cells mediates TAP1-dependent escape from cytotoxic T lymphocytes.Academic Article Why?
Lysophosphatidic Acid Is an Inflammatory Lipid Exploited by Cancers for Immune Evasion via Mechanisms Similar and Distinct From CTLA-4 and PD-1.Academic Article Why?
Multi-Specific CAR Targeting to Prevent Antigen Escape.Academic Article Why?
McCarter, MartinPerson Why?
Clinical and experimental approaches to the pathophysiology of interleukin-18 in cancer progression.Academic Article Why?
Suppressing the suppressor: Role of immunosuppressive regulatory T cells in cancer surgery.Academic Article Why?
The clinical evidence for targeting human myeloid-derived suppressor cells in cancer patients.Academic Article Why?
Fry, TerryPerson Why?
Torres, RaulPerson Why?
Slansky, JillPerson Why?
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